Chemistry Nobel Prize — DNA research lays foundation for new ways to fight cancer
My lab is interested in understanding how the DNA damage response is regulated specifically at telomeric DNA.
My lab is interested in understanding how the DNA damage response is regulated specifically at telomeric DNA.
While emerging anti-aging technologies are still emerging, the best-tested treatments we have for the present all rely on the insulin metabolism.
At first, only trivial amounts of beta amyloid begin to accumulate in small aggregates, but then they grow larger, gathering into huge amyloid plaques.
Mainstream evolutionary theory says that the body is forced to make compromises, and this this is the ultimate reason for aging.
There has been emerging a tradition by longevity researchers and activists around the world to organize events dedicated to promotion of longevity research on or around October 1 – the UN International Day of Older Persons.
Where we live – the air we breathe, the water we drink, the environments around us – has a huge impact on our health and even on our DNA.
Last year, I blogged on a CDC report that Alzheimer’s Disease is more prevalent than previous epidemiology had acknowledged.
RAMP2 can, under specific conditions, activate the molecules in the body’s cells that recognize the signal from the hormone glucagon. Were this to happen in the liver, it could promote the release of sugars into the blood -– precisely the process that it is supposed to prevent.
Tests with human subjects require decades, and are impossible to control, so the gold standard for testing claims for treatments that delay aging is the controlled trial with rodents, usually mice.
What would you say if I told you that aging happens not because of accumulation of stresses, but rather because of the intrinsic properties of the gene network of the organism?