Phase 3 Alzheimer’s drug increases toxic beta amyloid in the brain — but still provides benefits
Vienna, July 15, 2009 – New insights into how a Phase III Alzheimer’s drug might work were among the advances in potential therapies targeting two abnormal brain proteins – beta amyloid and phosphorylated tau – that were reported today at the Alzheimer’s Association 2009 International Conference on Alzheimer’s Disease (ICAD 2009) in Vienna.
Scientists also reported on how clinicians view and treat mild cognitive impairment (MCI), a research category used to define the state between normal aging and Alzheimer’s, that is now being used widely in clinical practice.
"There are now more than 5 million people living with Alzheimer’s disease in the United States. The cost of caring for people who now have Alzheimer’s, and those who will get it in the next few years, will bankrupt the healthcare system and devastate Medicare and Medicaid," said Ralph Nixon, PhD, MD, vice chair of the Alzheimer’s Association Medical & Scientific Advisory Council.